From this table, we can observe that convergence is achieved quickly, even in the DSRA case, where both the transmitter and receiver were adapting their antenna configurations.
Some Hfq-binding sRNAs act by binding to the coding region of mRNAs, as exemplified by MicC in Salmonella [35], or by binding to the 5'-region more upstream from the translational start site, resulting in activation of translation, as shown for DsrA in E. coli [36], [37].
This prompted us to investigate whether sncRNAs in addition to DsrA and GadY, were involved in AR; and if so, to establish how they exerted their regulatory actions.
A stenosis of at least one main renal artery was identified with intravenous DSRA in 22 patients and confirmed in 20 patients.
However, both the common themes (auxiliary metabolic genes (dsrC); some shared taxa) as well as differences (no dsrA in this paper; some differences in taxa identified) should be discussed.
In particular, for the measurement data, in the Distributed DSRA case, 26% of the scenarios did not converge before 10 iterations.
Notable examples include the sRNA RNAIII activation of hla in Staphylococcus aureus [ 52] and DsrA activation of rpoS in E. coli [ 53] (Fig. 2A).
Genes known to be involved in dissimilatory sulfate reduction including dsrA (codes for the sulfite reductase subunit [85]) were only observed in JCHS and CS.
Loss of YbeY resulted in up-regulation of several sRNAs (e.g. OxyS, DsrA, MicA, CyaR etc). in response to HU that in turn affect several cellular processes central to adaptation to oxidative stresses.
In particular, for the Centralized DSRA scheme, simulations show an increase of around 50% when using reconfigurable antennas, whereas for the measurements, the percentage increase is around 75%.
