Phenotypic plasticity refers to the ability of a given genotype to produce different phenotypes in distinct environments.
Alternatively, although some of the biological processes required for development through the pre-erythrocytic and erythrocytic cycles are similar, they occur in distinct environments, which necessitate the use of distinct sets of genes/protein to be carried out efficiently.
Jogler et al. [13] proposed separate horizontal gene transfer events could account for magnetotaxis in organisms like MC-1 and AMB-1 that live in distinct environments (marine vs. fresh water).
In such instances, opportunities for physical isolation in distinct environments provide ideal conditions that promote intraspecific diversification.
Phenotypic plasticity is the ability of a single genotype to produce different phenotypes in distinct environments [ 1- 4].
Differences in geographic distribution of serovars between ecological zones on Tutuila suggest that the three main serovars have different animal hosts that live in distinct environments.
On the other hand, diverse systems have been tailored to meet the differential metabolic demands in organisms living in distinct environments.
DeLong et al. (2006) sequenced large-insert libraries derived from microbial communities sampled at different depths in the North Pacific Gyre, and noted the enrichment of particular gene categories in distinct environments, which they hypothesized to reflect distinct adaptive strategies.
Adaptive Aneuploidy in Cancer Cells: Single clone cancer cells derived from an aneuploid cancer subpopulation are capable of adapting their aneuploidy rate and display distinct aneuploid rates in distinct environments, namely primary and metastatic microenvironments.
Furthermore, they refer to literature asserting that single clone cancer cells derived from an aneuploid cancer subpopulation, are capable of adapting their aneuploidy rate, and they display distinct aneuploid rates in distinct environments, namely primary and metastatic microenvironments.
Accordingly, the ability of E. dispar to become virulent under certain conditions may be due in part to the possible change of location of Prx in distinct environments [ 114].
