Oncogenic Met knock-in mice developed a spectrum of mammary cancers (solid adenocarcinomas, adenosquamous carcinomas, and myoepitheliomas), with some of them displaying histological, cytogenetic, and phenotypic characteristics typical of basal-like cancers, such as cytokeratin 5 expression and absence of progesterone receptor and Her2 expression.
The authors in [6] consider the stochastic characteristics of spatial and temporal spectrum availabilities in CRNs and develop a spectrum handoff scheme, which selects the handoff channel offering the maximum capacity.
Mutations in such susceptibility genes result in familial predisposition to developing a characteristic spectrum of diseases (collectively known as Hereditary Cancer Syndromes).
Thus, Bph3 has great potential in developing broad spectrum and durable resistant varieties against BPH.
The interest in developing new spectrum utilization technologies – combined with both the introduction of software-defined radios (SDRs) and the realization that machine learning can be applied to radios – is creating intriguing possibilities for promising technologies that are being incorporated in CRs. Figure 1 shows the overall behavior of a CR system.
In order to develop a design spectrum, the vibration properties of the structure is assessed that is natural frequencies, natural modes and modal damping ratio.
Mutations in the TP53 tumor supressor gene usually occur within the highly conserved DNA-binding domain (aa 100 298) and, when inherited, are typically associated with Li-Fraumeni syndrome (LFS), in which carriers develop a broad spectrum of cancers (e.g., breast, brain, soft tissue, bone, blood, and adrenal cortex tumors) during childhood or young adulthood [ 1].
Association between genetic elements and leprosy susceptibility has been suggested previously.[14], [15] Leprosy develops in a spectrum of clinical forms, from self-healing to the chronic lepromatous type of leprosy.
In this section, we develop a decentralized spectrum allocation and partitioning scheme.
Here we show that mutant mice in which SRF is selectively ablated in adult post-mitotic myofibers develop a broad spectrum of alterations similar to those which have been described in age-advanced skeletal muscles.
