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They may present clinically as localised pain and swelling, a pulsatile haematoma [4, 5, 6], compartment syndrome of the thigh [7] or compromise of the distal perfusion in case of peripheral occlusive disorders.
In case of peripheral PsA, RA controls were accepted.
In case of peripheral anterior synechiae with permanent angle closure, mydriatics and cycloplegics may be contraindicated.
In case of peripheral nerve injuries with long distance defects (gaps), autologous nerve grafting is the treatment of choice.
In case of peripheral neuropathy or oedema (grade 2) or gastrointestinal toxicity (diarrhoea, nausea, and vomiting grade 3) treatment was delayed until recovery for a maximum of 2 weeks and patients were retreated with docetaxel 60 mg/m.
Following this ASAS statement, all patients had an active disease as defined by a bath ankylosing spondylitis disease activity index (BASDAI) of ≥4 (scale 0 10) [ 11], had failed to previous therapy with at least two nonsteroidal anti-inflammatory drugs and sulfasalazine in case of peripheral arthritis, and should be treated with TNF-blocking agents according to an expert.
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In intensive care, prophylactic platelet transfusion should be performed in cases of peripheral thrombocytopenia < 10 × 109/L.
In intensive care, prophylactic platelet transfusion should be performed in cases of peripheral thrombocytopenia < 10 × 10/L.
Yang et al. [ 30] showed a high predictive value of malignancy (79%) in cases of peripheral vascularity depicted with Doppler in palpable axillary lymph nodes.
Although microtubule stabilization is protective against degeneration in a number of models, overstabilization of microtubules may also be detrimental as seen in cases of peripheral neuropathy in cancer patients treated therapeutically with taxol [ 50].
Conversely, there is an increased thrombotic risk in case of a peripheral mechanism, such as platelet activation, which can occur, for example, in the presence of disseminated intravascular coagulation (DIC), frequently encountered in the ICU [8],[9].
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