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Interestingly, more than 50% of the genes involved in adhesion contain CUG codons.
There, the switching in adhesion is achieved by a reversible buckling of adhesive pillar structures.
This tendency in adhesion persisted for all the test periods.
Changing the content of adhesion molecules could be responsible for the changes in adhesion force.
In adhesion mapping, the darker color referred to less adhesion performances and vice versa.
In adhesion and suspension fractions (A) and cell viability compare with control (B).
Some products reduce the formation of adhesions by affecting the biological processes involved in adhesion development.
Therefore, polysaccharides were also tested in adhesion assays (Fig. 5D).
PLC and MLCK signaling also play roles in adhesion, but do not exert NRG1α-specific effects.
In adhesion culture, ESCs primed to become osteoblasts recommitted to the adipocyte lineage in vitro.
Both YadA and Hia are important virulence factors involved in adhesion, invasion, and serum resistance.
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