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Taken together, we have shown that Wnt/ β-catenin signaling improves the proliferation of gastric CSCs in vitro and in vivo.
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While research also revealed that epithelium devoted to clear microbes away from mucosal surface in mounting responses and improve the proliferation of intestinal probiotics.
Importantly, when applying these polymers for myotube formation, the HPLAAT significantly improved the proliferation of C2C12 myoblasts in vitro compared to HPLA.
Cell culture assays in low serum culture medium (LSCM) demonstrated that incorporating very small amounts of aFGF and bFGF into the (Hep/Chi n multilayers significantly improved the proliferation of the NIH-3T3 fibroblasts.
The results indicated that moderate shear stresses (0.02 0.19 Pa) generated in the bioreactor improved the proliferation of rBMS cells to around 1.3 times of the static control with well maintained calcium deposition ability of the cells.
These scaffolds also greatly improved the proliferation of rat pheochromocytoma 12 (Pcellscells, and significantly promoted their differentiation and neurite growth along the nanofibrous alignment in the presence of nerve growth factor (NGF).
Furthermore, it was shown by western blot assay that the culture of chondrocytes in microcavitary hydrogel could improve the proliferation of cells potentially by inducing the Erk1/2-MAPK pathway.
The present data reveal that EGF and HB-EGF improve the proliferation of PCMOs by superactivating MEK/ERK signaling.
Results demonstrated that this perfusion bioreactor improved the proliferation of CSCs in vitro compared with standard culture methods.
It has been shown that there was no cytotoxic effect of the silks and growth factors improved the proliferation of mesenchymal stem cells.
Thus, this finding confirmed that NIH 3T3 wounded cells that were treated with nutrient rich maltodextrin improved the proliferation of cells; therefore it showed better performance in the proliferation of cells as opposed to the untreated cells.
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