Sentence examples for important cell surface from inspiring English sources

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Subsequent analysis of C57BL/6 bg/bg (beige) mice, which lack natural killer function, demonstrates that levels of asialo GM1 in the splenic T cell population do not increase with age but remain at the level of 2- to 3-week-old normal mice, indicating that asialo Gm1 may be an important cell surface component in the generation of natural cell-mediated cytotoxicity.

Integrins, the most important cell surface receptors that mainly bind to ECM proteins, consist of two non-covalently associated transmembrane glycoproteins subunits, α and β (Janes and Watt 2006; Kuphal et al. 2005; Villard et al. 2006).

Alpha-dystroglycan (α-DG) is an important cell surface receptor with high affinity for the extracellular matrix (ECM) proteins laminin, agrin, perlecan, and neurexin [1] and serves as a cellular receptor for several arenavirises, including lymphocytic choriomeningitis virus (LCMV) and the pathogenic Lassa virus (Cao et al., 1998).

TREM-1 has been identified as an important cell surface molecule involved in sepsis.

G protein-coupled receptors (GPCRs) are important cell surface signaling proteins with characteristic seven membrane-spanning (7TM) helices.

CD44 is a transmembrane glycoprotein which functions primarily as an important cell surface adhesion molecule interacting with hyaluronan [ 5].

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Bio-X affiliate Peter Parham examined a particular version of a gene which encodes for an important cell-surface protein that's a key player in our immune response.

An important cell-surface protein is CD4, the primary receptor of HIV-1.

Lipo-oligosaccharide (LOS) is one of the most important cell-surface structures expressed by C. jejuni, and strains associated with neuropathies express ganglioside-like LOS structures [ 15, 16].

Our proteomic resource also contains information on the differences in expression of important cell-surface ligands and receptors that can be mined to understand lineage development, differentiation and reprogramming.

In an in vitro mouse model of embryonic development, inhibition of autophagy prevented the upregulation of two important cell-surface engulfment signals – phosphatidylserine and lysophosphatidylcholine – in cells destined to die (Ref. 101).

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