Although these association are quite modest, the largest representing a mere 20 g sucrose/day per allele which corresponds roughly to 7 pieces of sugar or about one can of soda, they still implicate a relationship between GHRL genetics, sucrose and alcohol consumption, specifically in males.
Several lines of evidence implicate a relationship between attention-deficit hyperactivity disorder (ADHD), circadian rythmicity and sleeping disturbances.
However, the rare instances of multiple associations for individual SNPs implicate a relationship between the targeted genes (Supplementary file 2).
Emerging evidence strongly implicates a relationship between glutamate, NMDA activation and reactive nitrogen species in the pathogenesis of MS and the related model, EAE [1,9,46].
Several lines of evidence implicate a relationship between induction of SODs in tumour cells and reversion of neoplastic transformation or loss of the malignant phenotype including metastatic property.
Likewise, the association of leptin and IL-6 adds another tier to the wealth of data implicating a tight relationship between these cytokines, in particular highlighting how basal leptin may modulate subsequent IL-6 levels.
Interestingly, four out of the five putative MuERVs (red triangle) identified using the U3 probes from burn mice (K-4-11, U-4-8, and U-4-11) clustered into a unique branch (III), implicating a close evolutionary relationship.
Several studies have implicated a close relationship between high VEGFR2 expression in tumours and invasive and metastatic behaviour, increased proliferation and poor patient prognosis [3 6].
Our findings corroborate other studies that implicate a negative relationship between species diversity and hantavirus prevalence.
