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A variety of hormone axes have been implicated in the control of growth in vertebrates.
GILZ has also been implicated in the control of apoptosis and cell survival in various cell types [11, 15, 16].
The myoD family of DNA binding proteins has been implicated in the control of myogenesis in a variety of organisms.
A gene implicated in the control of protein degradation is mutated at high frequency in a metastatic eye cancer.
Acropetal transport of cytokinin (toward the shoot apex) has also been implicated in the control of shoot branching [6].
In animals, Mediator has been implicated in the control of differentiation and morphogenesis through modulation of numerous signaling pathways.
Genes that encode proteins implicated in the control of DNA transcription were downregulated in the intractable cancers.
LXRs are also implicated in the control of steroid metabolism.
Epigenetic changes have been implicated in the control of wound healing [14], [15].
The miR-2 family has been implicated in the control of apoptosis [23], [64].
Second, DLK1 has been implicated in the control of cell proliferation and differentiation [38].
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