Lethal (3) malignant brain tumor-like 2 (L3MBTL2), a novel polycomb group protein, has been implicated in regulating chromatin architecture.
Neuron-microglial signaling including purinergic, fractalkine, and neuroregulin-1 signaling have been implicated in regulating the proliferation of resident microglia in models of peripheral nerve injury [58, 59].
Open image in new window Figure 2 The INO80/YY1 chromatin remodeling complex is implicated in regulating BCCIP expression in HeLa cells.
RhoA is also particularly implicated in regulating cardiomyocyte gene expression.
We note that various phytohormones have been implicated in regulating the floral transition [5].
Thy-1 expression has been implicated in regulating inflammatory responses in fibroblasts.
Recently, several miRNAs have been implicated in regulating the initial step in epithelial-mesenchymal transition (EMT).
Cam activity has previously been implicated in regulating the stability and proteolysis of other integral membrane proteins [54].
Natural antisense transcripts (NATs) have been broadly implicated in regulating gene expression and chromosome structure [29] [34].
Inhibitory circuits have been implicated in regulating cortical plasticity.
Chromatin structure is implicated in regulating gene transcription in stress response.
