Common bacterial organisms implicated in aspiration pneumonia include Streptococcus pneumoniae, Staphylococcus aureus, Haemophilus influenzae, and Enterobacteriaceae [ 1].
While doctors know it was bacterial, they don't know exactly which of the more than 80 microbial "bugs" implicated in pneumococcal pneumonia was the culprit.
Since choline-binding proteins (Cbps) on bacteria have been implicated in S. pneumoniae adherence to host cells, their involvement in the differential adherence of S. pneumoniae strains was investigated.
Therefore, further studies are required to elucidate whether KpST66_1945 is implicated in K. pneumoniae pathogenesis.
Factors such as MagA (responsible for the mucoviscosity that correlates with high serum resistance in vivo[ 33]), siderophore aerobactin [ 34], and cell surface-associated fimbriae [ 35] have been implicated in K. pneumoniae virulence.
In contrast to viral neuraminidase, bacterial neuraminidase has not been implicated in combined viral/bacterial pneumonia or post-influenza pneumonia [ 34, 37, 40].
Over the last decade, pathogens traditionally thought to be confined to the hospital, such as Methicillin-resistant Staphylococcus aureus and Pseudomonas aeruginosa, are now implicated in non-nosocomial pneumonia.
Additional amoeba-associated bacteria might be implicated in community-acquired pneumonia, including Legionella-like amoebal pathogens (9 ) and members of the genus Parachlamydia (10 ).
Ameba-associated bacteria other than L. pneumophila, including other Legionella spp., new α-proteobacteria belonging to the Bradyrhizobiaceae (Bosea massiliensis) family, and members of the genus Parachlamydia might be implicated in hospital-acquired pneumonia (4 – 6 ).
While a number of organisms have been implicated in EE, Klebsiella pneumoniae has been recognized as an increasingly prevalent cause of EE in the Asian population [1, 2, 4 12].
