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Symptomatic disease can give rise to substantial pain, impairment of hand function and quality of life, leading to significant socioeconomic cost.
Finally, no systematic inventory was made of individual risk factors, such as ergonomics of the work place, subjective psychological distress, or underlying health disorders causing impairment of hand function.
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In consequence, many patients sustain impairments of hand function persisting even many years after the initial trauma [3].
Moreover, ongoing chronic pain accounting for increasing fear of movement, might similarly contribute to the measured level of hand function impairment.
Furthermore, as stated by Friedel et al. even a minor loss of hand function may lead to emotional consequences accentuating the need for outcome measures assessing more than physical impairment [ 23].
The loss of hand function is devastating.
Numerous studies have since demonstrated abnormal ipsilesional upper limb function when performing skilled motor tasks, irrespective of whether the lesion is cortical or subcortical, and currently there is no consensus on what types of deficit lead to impairment of ipsilateral hand function, independent of the site of the lesion (Nowak, et al., 2007; Winstein and Pohl, 1995).
Further tests in clinical subpopulations (eg, stroke patients) will determine if this variable might be a useful indicator of pathological impairment of proprioceptive hand function.
These contractures can lead to severe impairments in hand function[ 1].
This measure is reserved for a failure of treatment when residual proximal interphalangeal joint contracture is persistent and severe enough to cause serious impairment of digital motion and hand function.
Impairment of joint function in patients with hand OA is of great concern because of its impact on many activities of daily living and almost every type of employment.
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