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In addition, we collected information of serial image studies using sonography, magnetic resonance imaging (MRI), and computed tomography (CT), which included tumor size and location.
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Imaging studies using functional magnetic resonance imaging (fMRI) in human observers inspired yet another type of model for the interaction between attention and contrast.
Imaging studies using PMAP-1 and MitoAP-1 were limited to cultured cells [10] and T cells [19] until Bao et al. [20] first reported imaging studies of human PMNs using PMAP-1.
Imaging studies using FRET-based sensors have shown that cAMP levels in individual cells follow those of the surrounding cells due to diffusion through these junctions[20], [21].
Imaging studies using phosphorous magnetic resonance spectroscopy (31P-MRS) and 1H- MRS demonstrated reduced mitochondrial originated high energy phosphates, such as ATP and phosphocreatine (PCr) as well as other cellular factors whose metabolism is strongly suggested to be linked to mitochondrial ATP production, in schizophrenia relevant brain structures of schizophrenic patients [1] [8].
Imaging studies using a range of techniques have shown the relationship between tumour blood flow and glucose metabolism to be highly variable [17 28] (Table 1).
Imaging studies using this approach have reported so-called physiologic uptake in the FOM and other muscles involving the head and neck region.
Imaging studies using PET found that higher cortisol levels in response to amphetamine administration (Oswald et al. 2005) or to a psychosocial stressor (Wand et al. 2007) were positively associated with amphetamine-induced dopamine release in the ventral striatum.
Imaging studies using traumatic scripts have demonstrated that patients with dissociative PTSD have the opposite response pattern from patients with the reexperiencing/hyperarousal PTSD (as described above in "Flight or Fight in Humans").
Imaging studies using intravital microscopy in dermis tissue suggested that Ly6Clo cells scarcely leave blood vessels without inflammatory stimuli, but rather showed a patrolling behaviour rolling along endothelia in a LFA-1 and CX3CR1 dependent fashion [ 17].
Imaging studies using positron emission tomography (PET) in humans have corroborated the animal findings that drugs of abuse alter mesolimbic dopaminergic activity and have helped elucidate potential neurobiological underpinnings of drug addiction (for a review, see Martinez and Narendran 2009).
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