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With such sample, the transitional mechanism from form II to form I modification is further investigated by in situ WAXD combined with FTIR spectroscopy.
Surface engineering was carried out on oxide ceramics (Al2O3 ZrO2, ZrO2) using two different laser-assisted processes: (i) modification of the edge layer of the ceramic by laser induced remelting and alloying and/or (ii) surface microtexturing by laser ablation.
Under ambient conditions, there are two possible ways of such influence: (i) modification of nanocrystal surface leading to the increase of luminescence intensity and (ii) photooxidation of nanocrystals resulting in degradation of the luminescence spectra.
This increased intensity indicates increased collagen I modification around the invasive lesion [ 15].
Nevertheless, the HsdS proteins can combine with the HsdM proteins to form a functional type I modification enzyme.
The base specificity of Type I modification enzymes, i.e. m6A methylation as opposed to m4C and m5C of McrBC, is consistent with this hypothesis.
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In class I modifications, the saddle was designed as 10 mm mesiodistally, 2.5 mm buccally, and 2.5 mm lingually from the center of the implant (Fig. 2).
No quantitative or qualitative topoisomerase I modifications were observed.
Applying Phase I modifications on this substructure generates an arene oxide PCB3 (PCBI1) and 3-hydroxy-PCB3 (Fig. 6 PCBI4).
Accordingly, we used multi-photon and second harmonic generation (SHG) imaging to investigate collagen I modifications around invasive DCIS lesions in our model.
These are (i) modifications of interactions between existing proteins and (ii) the introduction of new nodes and links through gene duplications.
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