You could hypothesize, I suppose, that after the Medicaid expansion more people went to ERs for life-threatening situations and fewer for routine problems, and the two trends cancelled each other out — but then you would expect the hospitalization rate to rise even if the ER visit rate did not, because the visits themselves would reflect more serious conditions.
Comparable to that dichotomy, we could hypothesize that the two components of morality, i.e. emotion vs. cognition, are generally demanded differently, depending on the predominant moral concept of a person.
Endurance exercise, specifically marathon running, is a potent immune cell proliferative stress [ 87] and causes skeletal muscle remodeling (i.e., proliferation of muscle precursor cells [ 88]); thus, one could hypothesize that telomere length changes could occur following marathon running.
Based on the comparative genomics analysis alone, one could hypothesize that FrRUNT is simply a derivative form of RUNX2, i.e. the ortholog of zebrafish RUNX2A.
To explain these AI frequency differences between tumors with or without preoperative treatment, one could hypothesize that the clones that were sensitive to radiochemotherapy had a high level of alterations, i.e., a high number of altered chromosome loci.
Accordingly, one could hypothesize that the complex gets disrupted, and/or malfunctions, if Dpl does not interact with PrPC (i.e. in the absence of PrPC) thus reversing the beneficial effect of a neuroprotective signal.
We could hypothesize further that adding an extra feature will stimulate a competitor to match it.
One could hypothesize that non-synonymous variants with possibly larger effects might also contribute to this comparatively more severe phenotype.
We could hypothesize that an optimal way to perturb the global transcriptome, thus mimicking the virus effect would be to alter transcriptional hubs.
