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As increased HR activity of XRCC4 m/m fibroblasts during the cell cycle G2 phase involved a very small fraction of cell population, we hypothesized the activation of other compensatory mechanisms.
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It is hypothesized that the activation of the inflammatory cascade (via receptor activator of nuclear factor κ-B ligand [RANKL] signaling pathway) at the onset of acute Charcot neuroarthropathy leads to the activation of osteoclasts and subsequent bone and joint destruction (3– 5).
It is hypothesized that the activation of NF-κB and the subsequent expression of NF-κB-dependent genes is involved in the cellular response to components of the cosmic radiation.
More precisely, we hypothesized that the activation of the insular cortices during a visual experience of a loved one's pain would differ according to group.
Similarly, previous studies hypothesized that the activation of the A1AR may also be a potential cause for bradycardia in patients following cardiac transplantation.
Based upon the results of our comparative expression analysis, we hypothesized that the activation of a regulator(s) of heat shock/oxidative stress response inhibits TOR function and/or signaling.
We hypothesized that the activation of mTOR-associated signaling molecules may predict the efficacy of everolimus in mRCC patients.
We hypothesized that the activation of the EGF receptor and FAK, which results following C. jejuni infection due to the activation of β1 integrins, induces c-Src activity and the phosphorylation of caveolin-1.
We hypothesized that the activation of IKKα-dependent NFκB noncanonical pathway could drive the resolution of inflammation, as well as subsequent tissue regeneration or repair during the recovery phase of AKI.
In this context, it has been hypothesized that the activation of STAT-3 may be due to kinases that cannot be suppressed by SOCS-3 or that tumor cells may develop strategies to bypass the negative regulation mediated by SOCS-3 [ 40].
Therefore, we hypothesize that the activation of Treg population of the ceca is responsible for the enriched functional GO activity of lymphocyte activation and lymphoid organ development that was observed.
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