Since the Atg13 HORMA domain is similar to C-Mad2 [ 45], we hypothesized that Atg101 may potentially be a HORMA domain protein as well, perhaps capable of forming either an O-Mad2 state or both O-Mad2 and C-Mad2 states.
Defects in AID and UNG2 have been shown to result in the primary immunodeficiency hyper-IgM syndrome, leading us to hypothesize that additional, potentially more subtle, DNA repair gene variations may underlie the clinically related antibody deficiencies syndromes IgAD and CVID.
We hypothesized that independently created and maintained rule logic would potentially assess the patient's EHR data in different ways.
We hypothesized that older adults with CKD were at risk for potentially inappropriate treatment with metformin.
We therefore hypothesized that inhibition of PDE4 could prevent inflammation and hence the subsequent alveolarization impairement, and potentially oxygen-induced mortality.
We hypothesize that bacterial behavior potentially resides in other parts of the genome that were overlooked by previous studies, such as the so-called intergenic regions.
Extending this observation to other CNS cell types enables one to hypothesize that perforin could potentially deliver inflammatory factors to CNS cell types, including cellular components of the neurovascular unit, without initiating apoptosis.
This facilitates us to hypothesize that B. cenocepacia can potentially generate rhamnolipids.
We hypothesized that, despite potentially worse exertional dyspnea and cost of breathing, increased BMI in association with COPD would be associated with preserved constant workrate exercise time because of lower resting hyperinflation, which provides a mechanical advantage on respiratory mechanics.
It has been hypothesized that these potentially harmful practices have fostered behavioural problems in children and according to Smith and Mosby [ 14], create a vicious cycle of violence and the perpetuation of punitive, authoritarian parenting.
