Exact(3)
This concept, sometimes termed "the child survival hypothesis," was discussed and researched considerably, particularly during the 1970s.
A similar hypothesis was discussed earlier as a result of Wang et al (2002) finding association of the arginine allele exclusively in familial prostate cancer and not in sporadic disease.
In addition, Klaunig et al.'s claim that we (Keshava and Caldwell 2006) misconstrued their review (Klaunig et al. 2003) as focusing on DNA damage as a possible contributor to the MOA is incorrect; that hypothesis was discussed by Reddy and Rao (1989).
Similar(57)
This disparity with our hypothesis is discussed later in light of rickettsial signatures and the nature of Holosporaceae (see Discussion).
This hypothesis is discussed later.
Possible reasons to support this hypothesis are discussed.
The "Convergence hypothesis" is discussed as a possible explanation of such a trend in the conclusion.
Numerous implementation details are presented and the relevance of some modeling hypothesis is discussed.
A few pitfalls associated with the Anfinsen's thermodynamic hypothesis are discussed.
This novel mechanism of autotropic re-bending and its implications for the Cholodny-Went hypothesis are discussed.
This proposed hypothesis is discussed from connectional as well as developmental, pathological and chemo-architectural points of view.
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