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Our analyses support previous hypothesis of loss of acontia within taxa living in chemosynthetic environments [19], [44].
Our results revealed that the proportion of species bearing multiple dispersal syndromes is not lower among endemics (adapted to new island conditions) than non-endemic species (more related to mainland colonizers), as predicted by the historical hypothesis of loss of dispersability (see Vargas 2014).
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The evidence for immunoselection is least clear for DFTD, where the alternative hypothesis of loss-of-diversity in the host is tenable.
This is a well-known but unavoidable limitation of large epidemiological studies that may bias toward the null hypothesis because of loss of cases that presumably had more extreme values for the analyzed variables.
Therefore, we rejected this alternative hypothesis of identity loss in evolution, in strong support of the conclusion of the de novo origination.
Using the data sets in Arabidopsis, we tested the mutational-hazard hypothesis of intron loss and deduced its influence on the mechanism of genome reduction.
Thus no convergence phenomenon occurs within these phylogenetic groups, and the hypothesis of independent loss for small free-living cannot be rejected.
Considering the marked conservation of this cluster compared to the rest of the L. taiwanensis genome, the hypothesis of its loss in the rest of the analysed strains is very unlikely.
One major developmental hypothesis of nematode loss of both Myc and E CG) sites in RiBi core promoters might stem from the relatively small number of 1000 somatic cells in the adult nematode [ 40].
The "in-depth" similarity search allowed us to check for possible traces of these gene family members in other species and to support the hypothesis of actual loss in mouse and human genomes.
So there seems to be no trend in parasites and symbionts in general, an observation that suggests extra caution with regard to the selective hypothesis of TAS loss in intracellular microbes.
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