Sentence examples for hypothesis of lacking from inspiring English sources

The null hypothesis of lacking phylogenetic dependence was rejected (P = 0.05) for all 59 codon variables with Abouheif's Cmean statistic for DENV1 3 (Table S5).

However, although the phylogenetic dependence varied across the 59 codons, the null hypothesis of lacking phylogenetic dependence was rejected for all DENV1 4 by means of Blomberg's K statistic (Table S6).

Rather, similar to the reasoning underlying our hypothesis of lack of phylogeny-wide signal for soil variables, we suggest that phylogeny-wide statistics for ecological traits require a distinct interpretation.

Of the possible explanations for the increase in virulence observed here, we conclude that the hypothesis of a lacking trade-off between virulence and spore production is the most likely driver of the evolved increase in virulence.

Permutation tests revealed that the null hypothesis of a lacking phylogenetic signal cannot be rejected: the p-values for cranial and mandibular data were non-significant, either when using branch length estimates from Krause et al. [ 16] (pcranium = 0.67, pmandible = 0.49) or branch lengths set equal to 1 (pcranium = 0.66, pmandible = 0.49).

Robust experimental evidence in support of this hypothesis is lacking.

The predation of V. cholerae by phages following their co-ingestion from the environment is central to this hypothesis; however, molecular evidence in support of this hypothesis is lacking.

Maybe the hypothesis of a lack of wit and wisdom in today's Hollywood is not so far-fetched.

These data fail to demonstrate the hypothesis of the lack of specificity of the pharmacological tools used here, and in particular of the inhibitors of anandamide cellular uptake, hypothesis that instead would have been supported by the observation of identical sensitivity of PCL-NP-AEA and "free" AEA uptake to these compounds.

However, several arguments suggest that the hypothesis of the lack of antihypertrophic function of FHL2 mutants is unlikely.

This result supports our hypothesis of a lack of sensitivity of SAMP8 and SAMR1 mice, mostly attributable to their AKR/J genetic background.