Exact(20)
To make the public good hypothesis more plausible and move it beyond a speculative hypothesis, it would be useful to quantify the evidence in its favor.
However, according to this hypothesis, it would be expected that this signature be also activated in tumors, not specifically in NAT as we observed.
According to this hypothesis, it would be expected that, when studies analyse samples from different archaeological areas and periods, various types of binders will be identified.
To use the epidermal cells to test this hypothesis it would be necessary for them to express KRT1 mRNA and translate it into protein at a sufficient level.
To verify this hypothesis, it would be interesting to focus for instance on avrXv3 since it is altered by IS1595 in all tested pathovar alfalfae strains.
To test this hypothesis, it would be interesting to combine bioinformatics and transgenics production to identify the zebrafish KV dmrt2 enhancer and show that it is indeed absent from the mouse genomic sequence; 2) from the loss in the mouse of a protein(s) necessary to activate specifically the node enhancer.
Similar(40)
While we could come up with different hypotheses, it would be useful to investigate this process directly.
To address these potentially competing hypotheses, it would be useful for the authors to quantify responses in a slightly different ways to resolve these issues.
To examine such hypotheses it would be useful to establish the corresponding receptor repertoires of aquatic species from the tetrapod lineage.
Clearly, to support any of these hypotheses, it would be of enormous benefit to generate high-resolution structural data to identify exactly where in the mitoribosome ICT1 is found.
The suggestion is that when this hypothesis holds, it would be morally inappropriate to insist on maintenance of equal opportunity.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com