However, some obese persons are unable to produce sufficient amounts of insulin, and thus the compensatory increase in response to increased blood glucose concentrations is inadequate, resulting in hyperglycemia.
Other patients present with symptoms of hyperglycemia that have been present for months or with the sudden onset of symptoms of very severe hyperglycemia and vascular collapse.
In overweight or obese patients with type II diabetes, caloric restriction for even just a few days may result in considerable improvement in hyperglycemia.
Mild hyperglycemia causes no symptoms, but more severe hyperglycemia causes an increase in urine volume and thirst, fatigue and weakness, and increased susceptibility to infection.
Diabetes mellitus is a group of metabolic disorders of carbohydrate metabolism characterized by high blood glucose levels (hyperglycemia) and usually resulting from insufficient production of the hormone insulin (type 1 diabetes) or an ineffective response of cells to insulin (type 2 diabetes).
Most patients present with symptoms of hyperglycemia, but some patients present with diabetic ketoacidosis, a clear indication that insulin secretion has significantly deteriorated.
Elevated blood glucose (hyperglycemia), as can occur in diabetes, is also dangerous and cannot be left untreated.
A glycosylated hemoglobin called hemoglobin subtype A1c (HbA1c) is particularly useful in monitoring hyperglycemia and the efficacy of diabetes treatments.
There are two acute life-threatening complications of diabetes: hyperglycemia and acidosis (increased acidity of the blood), either of which may be the presenting manifestation of diabetes.
This has a variety of physiological consequences, including hyperglycemia (high blood glucose levels), which can lead to type II diabetes mellitus, and hypertension (high blood pressure), which can lead to cardiovascular disease.
When food is eaten, there is a temporary rise in the blood glucose level known as alimentary hyperglycemia (high blood glucose level).
