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rCBF was measured in both parietal cortices concurrently by the hydrogen clearance.
Intrinsic blood flow near the stump tip, examined using microelectrode hydrogen clearance polarography was highly nonuniform and included zones with very low perfusion.
To estimate changes in cerebrovascular responses to hyperoxia, CBF was measured by hydrogen clearance in anesthetized rats, either control animals or those pretreated with one of these blockers, with the NO inhibitor N omega -nitro-l-arginine methyl ester (l-N omega -nitro-l-arginineS-N omega -nitro-l-argininemiN omega -nitro-l-arginineker comethyl with l-NAMester
Tumour blood flow and the change in tumour blood flow induced by AC7700 were measured by the hydrogen clearance method.
To investigate whether tumour vessels or normal vessels are more sensitive to AC7700, we constructed a new type of wire electrode for use in the hydrogen clearance method.
TBF was measured with the hydrogen clearance method (Hori et al, 1991, 1996) whose principle was described in detail by Aukland et al (1964).
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TBF was measured by use of the hydrogen-clearance method (Hori et al, 1991, 2002) that was originally developed by Aukland et al (1964).
Previously, a computational model of cellular hydrogen peroxide clearance was developed and optimized for wild-type Jurkat cells [ 1].
The dynamic analysis of intracellular hydrogen peroxide clearance indicates that surprisingly, while perturbing Trx1, Grx1, and G6PD have large consequences on antioxidant capacity, additional non-specific effects likely only modify the redox capacity of Grx1 shRNA transduced cells.
In prior work, we have quantitatively described through computational modeling the collective properties of the redox enzymatic network as it pertains to exogenous hydrogen peroxide clearance from the cytosol and protein thiol oxidation/reduction [ 1].
A previous study has also shown that KRG improves the systemic blood flow, such as liver, spleen, kidney, and gastric mucosa in rats, using the hydrogen gas clearance method [ 26].
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