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As humans we find safety in numbers but we also want to differentiate ourselves.
As humans, we find ways to cope -- for some these things are positive, and for others they are negative.
In humans we find that kinetochore binding by the hNDC80 complex occurs independently of the hSPC105 complex, confirming previous reports [15], [16].
In humans, we find highly expressed miRNAs to be biased towards the 5p hairpin arm.
In humans, we find here an inverse correlation between drinking water lithium concentrations and all-cause mortality in 18 neighboring Japanese municipalities with a total of 1,206,174 individuals (β = −0.661, p = 0.003).
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In humans, we found strongest activation to the same stimuli in areas V3A, V7, the V4d topolog (V4d-topo), and a caudal parietal disparity region (CPDR).
When we compared our bat DNA sequences with those obtained from goats, pigs, cows, dogs, chickens and humans, we found that Diphylla had consumed blood from chickens and humans.
However, for the efficient infection of mice and probably other mammals including humans, we found that a lower pH optimum of the HA activation might be required.
However, in contrast to the robustly observed face-evoked N170 component in humans, we found that faces did not elicit a peak in the latency range of 150 200 ms in either experiment.
In constrast to humans, we found that the proportion of peripheral memory B-cells was higher than that of naive B-cells (68±4% versus 11±2% of B-cells, respectively) in non-infected macaques (Figure 1.C).
Moreover, in humans, we found that different tissues use distinct classes of promoters, reflecting an emerging promoter network.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com