Your English writing platform
Discover LudwigExact(4)
Van de Steeg et al. reported that complete OATP1B1 and OATP1B3 deficiency caused human RS by interrupting conjugated bilirubin reuptake into the hepatocyte [12].
ROR2-deficient mice exhibited severe skeletal defects which are analogous to those of human RS mutations including dwarfism as well as heart and lung malformation [6], [7].
Thus Prickle1 hypomorphs recapitulate most phenotypic features of human RS patients (Butler and Wadlington, 1987; Patton and Afzal, 2002).
In the present study, we show that null and hypomorph Prickle1 mutations in mice manifest clinical features of human RS caused by mutations of Wnt5a pathway.
Similar(56)
Lowe, D. G. & Goeddel, D. V. Heterologous expression and characterization of the human R-ras gene product.
This same third component may also exist in the human Red nucleus.
Full-length human R-Ras was cloned into EcoRI and SmaI-sites of pBTM116 to yield BTM116-R-Ras.
Lastly, bovine reassortant viruses that had inherited a rearranged segment 7 from human r-RV were generated.
In the present study, we have sought to better delineate the morphological determinants of the adult human Red nucleus (ahRn).
For the sake of illustration we have applied this approach to the human Red blood cell metabolism (hRBC) and its capacity to predict temporal phenomena was evaluated.
In this study, results obtained from one-step viral growth curves indicate that human r-RV replicate less than or equally to their normal counterpart.
Write better and faster with AI suggestions while staying true to your unique style.
Since I tried Ludwig back in 2017, I have been constantly using it in both editing and translation. Ever since, I suggest it to my translators at ProSciEditing.

Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com