Sentence examples for human prostate cell from inspiring English sources

The biological effects of these compounds on three α1-adrenoceptor subtypes and cancerous human prostate cell lines (PC-3, DU-145, and LNCaP) were determined.

We exposed two human prostate cell lines (LNCaP clone FGC and PNT1A) to nutritionally relevant doses of MSC and selenite, ranging from deficient to the equivalent of selenium supplementation in humans.

PpIX kinetic measurements and flow cytometry show accumulation of PpIX upon incubation with phosphatase-sensitive prodrugs in PC3 human prostate cell cancer, MCF7 breast adenocarcinoma, U87MG glioblastoma, T24 bladder cancer and A549 lung carcinoma cells.

This work was designed to determine whether IGF-1 and EGF modulate nuclear transfer and transactivation of the androgen receptor (AR) in human prostate cell lines (PNT1A and DU-145).

To investigate the molecular function of HSF2 in PrCa, we analyzed the mRNA expression in a panel of human prostate cell lines with different malignant potential and invasive behavior.5 HSF2 levels were very low in basal and non-transformed prostate epithelial cells (PrEC and EP156T) and high in malignant PrCa cell lines of the luminal type (LNCaP and Du145) (Figure 2a).

A representative compound (30w) showed potent p21 promoter activity, comparable with that of trichostatin A (TSA), and its cytostatic activity against cells of the human prostate cell line LNCaP was more potent than that of the well-known HDAC inhibitor, suberoylanilide hydroxamic acid (SAHA).

Here, to find novel genes, we analyzed all the transcripts expressed in normal human prostate cells using this microarray.

KBU2046 inhibits cell invasion with efficacy equal-to-or-greater-than that of genistein for human prostate cells, including normal prostate epithelial cells, as well as primary and metastatic PCa cells (Fig. 1b).

It was not toxic to human prostate cells (Table 1), to human bone marrow stem cells (Fig. 1d), nor to cells in the NCI-60 cell line panel (Supplementary Fig. 2).

She is analyzing its effects on the prostate in young mice and rats, and also in rats that have been implanted with human prostate cells.

The treatment of androgen-independent DU145 human prostate cells with a 21-mer MMP-9 antisense PMO caused a dose-dependent inhibition of cell proliferation compared to scrambled or MMP-2 antisense PMO at similar concentrations.