We cannot however exclude that DNA methylation changes at single or selected CpGs within promoters may lead to transcriptional perturbations as the MeDIP-array approach is likely not sensitive enough to detect very discrete alterations.
We cannot, however, exclude that treatment with various potentially effective medications may have influenced our findings.
We cannot however exclude that our observations are due to the induction of other unknown proteins.
We cannot, however, exclude that this case is in a preclinical state preceding amyloid deposition detectable by PET [ 14].
We cannot, however, exclude that accumulation in the ER in secondary to the overexpression of these proteins.
Based on our data and with reference to previous studies we cannot, however, exclude that in rare cases identical copies with different splicing patterns may exist.
We cannot, however, exclude that the increased expression of TLR2 by A549 cells is solely and directly caused by cyclic stretch.
We cannot, however, exclude that some TS cells directly switch from parental to inverted I-XCI upon HAT selection without transiting through a "two-active-X" state.
We cannot, however, exclude that HERB-1 still infects some localized, genetically unimproved host populations, since we have explored only a fraction of current P. infestans genetic diversity.
We cannot, however, exclude that differences in age, smoking habits and comorbid disorders could contribute to the observed differences in levels of tryptophan and kynurenines.
We cannot, however, exclude that the variant may act as a modifying or low penetrance gene that may exhibit incomplete segregation and retention of the wild-type allele, as has been demonstrated for the CHEK2 gene [ 9, 10].
