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Take oil prices, for example: these (and government subsidies) determine how much maize is planted for ethanol.
Before these biomarkers, the primary way to estimate aflatoxin exposure was to observe how much maize and nuts people consumed on average and to measure or assume aflatoxin levels in these foods.
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To estimate how much real world choice maize farmers have in countries with different degrees of GM crop adoption (Austria, Germany, Spain, Switzerland), we used surveys of seed catalogues from local and regional seed suppliers, transnational seed corporations and public national and European seed registration catalogues as an approximation for real world choices available to farmers.
Hence another question of importance is the relative obesogenic effects of different grains, and how much impact minimally processed rice, maize, millet, sorghum, or sprouting grains have on metabolism and microbiota.
If a perennial biomass crop like Miscanthus could be bred to produce maize-like grain, how much reallocation of existing production from stems to seeds might be necessary?
"a robust experiment would also include a random, unrelated diet, e.g., one derived from organic maize)….. Critical details on how much food was consumed by each rat are absent, making it impossible to establish any dose/response relationship".
Aflatoxin exposure is a function not only of aflatoxin concentrations in maize and nuts but also of how much of these foodstuffs individuals consume in different parts of the world.
So it is not clear how much the tumours were the result of feeding them GM maize and how much was spontaneous.
That in turn influences how much land is planted to soyabeans, which for American farmers are interchangeable with maize.
How much?
How much, how soon?
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