We used maximal 8-hour concentration of ambient ozone because WHO recommended that maximal 8-hour mean reflects the most health-related exposure to ozone [ 23].
The increase in cetuximab wastage rate during the second semester of 2006, which was probably due to a demanding 24-hour concentration of chemotherapy sessions for colorectal cancer patients, is an example of such a hindrance.
When we estimated the association between 24-hour concentration of PM2.5 and PEF, we observed a significant association in the single-pollutant model in both the morning and evening (-2.96 l/minute (95% CI: -4.55, -1.37) and -4.42 l/minute (95% CI: -7.11, -1.73) for a 24-hour mean PM2.5 concentration of 10 μg/m, respectively) (Table 4).
A Japanese panel study showed that the change in morning PEF for a 10-μg/m3 increase in the 3-hour concentration of SPM measured at 2 a.m. to 5 a.m. on the same day was -0.76 l/minute (p < 0.05) among children with asthma in April through September using a single pollutant model [ 4].
A total of 11 (N) variables including temperature (°C), amount of biomass (%, w/v), pH, time (hours), concentration of cellulase and xylanase from P. ostreatus and of commercial cellulase from Trichoderma reesei ATCC26921 (U/gds), and 5 unassigned variables (dummy) were studied in 12 (N + 1) experiments.
The judges rejected a challenge from the American Petroleum Institute, ruling that both science and the law justified a 2010 decision by the Environmental Protection Agency to limit maximum one-hour concentrations of nitrogen dioxide in the atmosphere to 100 parts per billion.
After 24 hours, concentrations of IL-6 and IL-1β were measured with an ELISA method.
We used moving averages of maximal 8-hour concentrations of ozone monitored at 8 stationary stations as personal exposure.
