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Finally, we tested whether host ethnicity, host gender, host age, or the cell wall phenotype of the infectious agent had an effect on the number of informational genes.
Host (gender, age, weight, transaminase levels, fibrosis stage, and source of infection) and viral-related factors (viral load, and genetic variability in the E1 E2 and Core regions) were assessed.
Structural variation across host gender: We further assessed microbial structure variation with respect to host gender.
It was, therefore, not possible to analyse host gender differences in parasitic burden and diversity.
However, these habitats perform different degree of structural variation with respect to host gender.
And skin communities had no unique structural variation patterns regarding to host gender.
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I examined uninfected males and females separately, because host genders may have different susceptibilities and, consequently, different allocation patterns.
Table 2 summarized the distribution of 1920 metagenomic samples on three body habitats and two host genders.
The clustering results indicate that structure of human microbiome is varied systematically across body habitats and host genders.
Next, from the clustering results, we infer how microbial pattern was influenced by body habitats and host genders.
The aim of the present study was to estimate the prevalence of Dirofilaria immitis infection, and to examine the relationship between host factors (gender, age and breed) and D. immitis infection in dogs.
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