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Histological analysis indicated that genome duplication regulated the root response to salt stress.
Histological analysis indicated a transformation into the SCC subtype (Kuiper et al., 2015).
Histological analysis indicated increased inflammation in the BD lung tissue, consistent with previous literature [1, 8].
Furthermore, the histological analysis indicated that the cells could adhere in modified silk fibroin scaffolds.
At 12 weeks, all the nerve conduits remained structurally stable without any collapse and histological analysis indicated axonal regeneration in the nerve conduits of both groups.
Histological analysis indicated greater tumor penetration, more extensive ECM remodeling, and greater cell destruction in tumors administered PTSL, correlating with improved response to the therapy.
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Expression of GPR126 could not be detected in typical cardiac neural crest (CNC) locations [20], and histological analysis indicates that septation and the formation of the CNC-derived outflow tract are initiated normally (Fig. 6).
Histological analysis indicates that angiotensin II-induced cardiac hypertrophy and fibrosis are significantly promoted in Fgf16 knockout mice.
Histological analysis indicates a mixture of adenocarcinoma, squamous cell carcinoma, and one large cell carcinoma in the cohort.
The results from the present study and histological analysis indicate the administration of Qurs Tabasheer, has significantly protective effects against STZ-induced diabetic state.
Histological and cellular analysis indicated that these tumours were CPCs resembling their human counterparts.
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