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Here we re-evaluate data supporting these roles in the context of mechanistic insights provided by recent genome-wide studies and highlight evidence for CTCF-mediated intra- and interchromosomal contacts at several developmentally regulated genomic loci.
When possible, we highlight evidence for features of human neocortical architecture and function that cannot be easily explained as correlates of brain size and, hence, might be more directly associated with the evolution of uniquely human cognitive capacities.
Here we provide a succinct account of the biology of CK2, its cellular substrates, its pro-survival and pro-proliferation activity, and highlight evidence for its involvement in human cancer.
Leveraging RNA sequencing from 636 human brains, we discover differential expression for several retrotransposons in association with neurofibrillary tangle burden and highlight evidence for global TE transcriptional activation among the long interspersed nuclear element 1 and endogenous retrovirus clades.
Next, we review recent literature on plant functional traits to highlight evidence for a fundamental trade-off between flowering phenology and the return rate of growth investments, which may impact how climate change and phenology affect invasions.
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This review highlights evidence for the roles of the major DNA repair pathways in mycobacterial pathogenesis and considers their potential identification as targets for novel compounds, especially those designed to inhibit the evolution of resistance.
This study has highlighted evidence for follow up strategies that are likely to improve survival and well-being of women treated for early breast cancer.
In this review we highlight evidence that provides support for deferring ASCT in favor of additional anti-myeloma therapy.
We critically highlight some evidence for the importance of soil biodiversity to sustaining (agro- ecosystem functioning agro- ecosystemections functioningreseandh.
We highlight recent evidence for effects of anthracyclines on DNA torsion and chromatin dynamics that may underlie basic mechanisms of doxorubicin-mediated cell death and suggest new therapeutic strategies for cancer treatment.
These data highlight new evidence for a cross-talk between estradiol (E2) and growth-factor-induced cytoplasmic signaling.
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Since I tried Ludwig back in 2017, I have been constantly using it in both editing and translation. Ever since, I suggest it to my translators at ProSciEditing.

Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com