Sentence examples for high level virulence from inspiring English sources

In contrast, strain 99-738 DP-B5 exhibits a high level virulence more characteristic of clade 2 strains, i.e., it causes skin infection, systemic infection, and death (Thiaville, P.C., et al., Infect. Immun., submitted).

Importantly, Ohashi et al. determined the 50% mouse lethal doses (MLD50) for representative strains of each subtype and found that strains from subtypes Kato, JG, Gilliam and Karp exhibit high-level virulence to mice, that from JP-2 intermediate-level virulence and those from JP-1, Kawasaki, Kuroki and Shimokoshi low-level virulence.

As expected, the ΔsecA2 single mutant of M. tuberculosis showed a high level of virulence in this experiment, as all animals died in less than 50 days.

A high level of virulence is likely to evolve when the transmission time (between the initial infection and the release of new infective cells from the host) is short, since rapidly reproducing strains of the pathogen outcompete the slow grower, thus utilising more of the host resources.

V. vulnificus M06-24/O M06-24/Oa typical clade 2 strain exhibiting a high level of virulence in the subcutaneously inoculated iron dextran-treated mouse model [ 23- 25].

However a rather high level of virulence is expected as the strains were isolated from chickens with severe clinical symptoms and MD lesions.

Similarly, 36.6% of strains belonged to the ST131 clone and showed a high level of virulence and resistance, which could play an important role in the epidemiological success of this sequence type.

Strains that do not cause disease in swine may continue to express high levels of virulence in other hosts, such as humans [49].

This is most noticeable if both cP and cH are small, where regular coevolutionary fluctuations occur, with the host cycling through low levels of resistance and the parasite cycling through high levels of virulence (Fig 3D).

Almost nothing is known about the biology of Pae on European horse chestnut, including the reasons for its apparently sudden emergence, the genetic factors contributing to its observed high levels of virulence on a woody host or its evolutionary relationships to other strains of P. syringae.

In theory, highly specialized parasites can evolve towards high levels of virulence ([ 40], but see also [ 41]).