Sentence examples for hf response from inspiring English sources

When the corresponding HF response is gained, a function that maps the LF outputs to HF outputs is constructed via LSSVR.

The gene expression of nearly all the network components involved in the promotion of inflammation and immune response is induced during the early phase and repressed during the late phase of the HF response, including NF-κB regulatory factors, interferon, cytokine and chemokine signaling molecules, acute phase response reactants and complement components.

Considering its critical role, we propose the model in which down-regulation of NEMO in the mid and the late phases of HF response could be the most important regulatory event controlling the shut-down of NF-κB driven inflammatory response at the switch point between stressed and pathological hepatic state.

The pathway interaction network for the HF response is typed by several key proteins including Pik3r1, Kras, and Ctnnb1.

For example, this lead us to the interactions with the insulin signaling pathways in the late HF response, which were not central in the network but appeared to be relevant because of enrichment of all involved pathways and the central role of insulin in this analysis.

MB, a medical epidemiologist with a particular interest in FHF, contributed to the Masindi EHF response as consultant for the Ministry of Health, and on two occasions (Watsa/DRC and Uige/Angola) to Marburg HF response activities as volunteer for Médecins Sans Frontières.

Other aspects of the metabolic control, such as amino acid metabolism and tricarboxylic acid cycle, are also upregulated during mid and late phases of the HF-response.

The suggested mechanism of the mutual repression of PPAR and NEMO/NF-κB regulators during their coordination of the transition from early to the late phase of HF-response remains to be confirmed by biochemical studies.

Potentially, differences between mouse strains with respect to inflammation intensity that we observed may not represent variations in the intensity of hepatic inflammation developing in each strain, but rather HF-response patterns that are temporally out-of-step between strains.

The first and largest module contains gene sets that are generally significantly upregulated during the early phase (day 3) and downregulated at the late phase of the HF-response (week 8, 12 and/or 16).

The genetic determinants of heart failure (HF) and response to medical therapy remain unknown.