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Here we propose that IN content determines nuclear import.
Here, we propose that filopodia stability directs cell migration and promotes cancer cell invasion.
Here, we propose that TJ strands consist of claudin protomers that assemble into antiparallel double rows.
Here we propose that evolutionary forces can help explain this paradox.
Here, we propose that Sig-1R is a pluripotent modulator with resultant multiple functional manifestations in living systems.
Here, we propose that even small alterations in microtubule dynamics can influence cell migration via several different microtubule-dependent pathways.
Here we propose that temporal prediction recruits adaptive and non-adaptive oscillatory mechanisms involved in establishing such an internal representation.
Here we propose that direct, sequence-specific interactions between GR and some κBREs are critical for transcriptional repression previously explained exclusively by tethering.
From the evidence collected here, we propose that Hb could have an important role in the processes that culminate in PD neurodegeneration.
Here, we propose that the process of sortase-mediated pilus polymerization follows an expose-ligate-seal mechanism (for details, see Fig. 7d and Discussion).
Here, we propose that inhibitory control can be instantiated by an integral signal derived from working memory, another key function of the PFC.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com