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The ripple-like structure is more pronounced for the smaller thicknesses, and the worm-like structure appears for the Pt thickness of 50 nm when the PLLA is heated (Figure 3).
As expected, significant N2O desorption was observed when substrates were heated (Figure 7a); in the interval from 40°C to 75°C, all materials reached the N2O concentration of the control.
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Most significance was responses to stress and heat (Figure 3A).
As a result, a strong BN phase XRD peak was observed after the heating (Figure 5 (iv)).
All of these genes showed induction of expression in response to heat (Figure 6A).
AtPhox2 was found induced by heat (Figure 5), AtPhox3 by cold and salt stress, and AtPhox4 by cold, osmotic and salt stress, as well as pathogen infection.
The same was done for the descriptors for Aδ mediated pain by pooling the ratings of punctate mechanical pressure and laser heat (Figure 3).
Correspondingly, subjective ratings of thermal comfort and sensation were higher during the match in the heat (figure 2).
RPE increased during each 10 min of effective play in both conditions; however, it was higher in the heat (figure 2; p<0.01).
Upon further heating (Figures 6 and 7), growth occurred mainly at the free surface.
If things were heated, you'd figure Lance would at least get up".
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