Sentence examples for heart development in the from inspiring English sources
We have also discovered that the N1 gene product is related to heart development in the Mexican axolotl [Cell Mol. Biol. Res. 41 (1995) 117].
While the majority of studies have focused on heart development in the Drosophila embryo, attention has recently focused on the structure and function of the adult fly heart as a model of human heart failure.
This is undoubtedly the case for later stages of heart development in the mouse where penetration of staining reagents into dense cardiac tissue can be problematic.
To date, most analysis of heart development in the mouse has focussed on qualitative comparisons of normal and mutant hearts, usually using selected 2D histological sections.
Although this protein is necessary for heart development in the embryo, it is usually not expressed in the adult cell [ 22].
Moreover, other populations contribute to the heart development in different times such as Isl1 expressing cells from the dorsal coelomic wall [ 131] or Tbx positive cells [ 132], inevitably resulting in mixed population of cells in various stages of maturity.
In summary, our results show that Tie-1 and Tie-2 have redundant roles in heart development in zebrafish and more specifically in the maintenance of endocardial-myocardial interaction.
22 In particular, 16 of these miRNAs targeted a series of genes involved in heart development and CHD, like miRNA-424 and miRNA-222, whose overexpression was correlated with the reduction of heart development associated genes NF1 and HAS2, suggesting that these miRNAs might be implicated in the abnormal heart development in TOF.
NKX2-5 gencodesodes a highly conserved homeobox transcription factor, which is essential to the heart development in embryos and cardiac function in adults.
These results raise the possibility that ADAMs 9, 17, and 19 contribute to heart development in humans and have implications for understanding the mechanisms underlying congenital heart disease.
Loss of PRH expression or loss of PRH repression activity in the endoderm blocks endogenous heart development in Xenopus embryos and ectopic heart induction.
