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Several studies have demonstrated alteration in motor control strategies in patients with spinal pain [ 36- 38].
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Several studies have demonstrated alterations in serotonin 5-HT2A (5-HT2AR) and glutamate metabotropic mGlu2 (mGlu2receptorsors in depression, but never in the same sample population.
Although studies have demonstrated alterations in immune system function in the critically ill, this may represent a transition along the spectrum of immunocompetency opposed to true immunosuppression.
Studies using postmortem human brain samples have demonstrated alterations in the levels of α7 nAChRs in the brains of patients with schizophrenia [16], [17] and Alzheimer's disease [18] [20].
Several recent studies [ 36- 40] have demonstrated alterations in the central sensorimotor processing in CRPS patients.
A handful of studies have demonstrated alterations in both rats and mice.
Some studies have demonstrated alterations in VMO-VL onset times in AKP patients following or during successful therapeutic interventions.
Since 1983, studies using transgenic animal models have demonstrated alterations in mitochondrial enzymes in the Alzheimer's disease brain.
Epigenetic, expression and protein analyses have demonstrated alterations of IGF/INS expression and protein levels in cancer tissues.
Several studies have demonstrated alterations in neuropeptide and neuropeptide receptor expression in PBS/IC and in animal models of bladder inflammation.
Our studies in healthy subjects who carry this variant have demonstrated alterations in the composition of the transitional and naïve B-cell pool.
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