Although ∆oxyR was shown to be important for the bacterial survival at an intermediate RH, this mutant was no longer important at low RH as this mutant had the same viability as the parental strain.
∆soxR had the same viability compared to the parental strain at all RH levels.
This is in agreement with the fact that senescent cells show the same viability as normal cells.
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As a point of comparison, a population containing an uncommon (p = 0.1) completely dominant allele that reduces viability by 1% has the same magnitude of DHW as a population where every mating involves 4th cousins (F ≈ 0.0009).
Result of a cytotoxicity assay showed that neutrophils first incubated with anti-TLR2 at 5 μg/ml, anti-TLR4 at 5 μg/ml, RAGE-Fc at 5 μM, MyD88 inhibitor at 20 μM or NF-κB inhibitor at 20 μM for 1 h on ice had same viability as those with buffer control for 1 h on ice.
First, PINK1 MUL1 and parkin MUL1 double null mutants show a high frequency of pupal lethality as compared with single mutants (data not shown), while double null mutants of PINK1 parkin have the same level of viability as single mutants (Clark et al., 2006; Park et al., 2006).
It was also noted that cells at 50 μg/mL of BCNU + 50 μg/mL GSH as well as at 25 μg/mL IFNQ + 25 μg/mL GSH had the same percentage viability at both 48 and 72 hours.
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