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HAPs data from 1990 , 1996 and 1999 were available.
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As discussed in "Methods," the U.S. EPA TRI data contribute to the NATA HAPs data used in our study; however, the NATA data are also supplemented by data from local air pollution inventories and other U.S. EPA air toxics databases (U.S. EPA 2011).
We geocoded the birth addresses to obtain census tract for linkage to HAPs data.
In this study, we estimated the cancer risks of Hispanics and non-Hispanic whites due to exposure to HAPs using data from the RIOPA study.
HAP data were downloaded from the U.S. EPA website on 23 June 2010, and additional archived data were received on compact disc from the U.S. EPA (2011).
RESULTS: Among 314,007 ischemic stroke patients at 1244 Get with the Guidelines-Stroke hospitals from 2003-2009 wereweligibleible for DS and had completed HAP data, a total of 216,372 (68.9%) underwent DS and a total of 17,906 (5.7%) developed HAP.
We linked the HAP data with the NHS, using U.S. census state, county and tract identifiers (U.S. Census Bureau 2010).
The Partek algorithm assumes that the pooled data from Hap Map 6.0 represent an average CN of 2. CN calls were made by comparing intensity values of each test sample to the pooled reference sample.
However, data from IMPACT-HAP were for the most part prospectively collected and validated before entry into the data repository.
Estimated ambient concentrations of hazardous air pollutants (HAPs) were assigned to CTS participants using data from the United States Environmental Protection Agency EPAA).
More recently, the U.S. EPA conducted the National Air Toxics Assessment (NATA) using emissions data from 1996 for 33 priority HAPs, most of them VOCs (U.S. EPA 2001).
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