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In the tVNS group, stimulation (0-10 V, 1 ms, 20 Hz) was continuously performed during 30 minutes, starting 10 minutes before intravenous administration of 2 ng/kg E. Coli LPS.
In this CCD-positive patient group, stimulation differed by up to two orders of magnitude between wild-type and GTI extracts (Figure 6C).
In the tVNS group, stimulation of the vagus nerve was achieved as indicated by laryngeal vibration.
In the healthy group, stimulation with flicker light induced a vasodilatation of 7.0 ± 2.3% in retinal arteries (ANOVA, time effect, P < 0.001) (Fig. 1) and a dilatation of 6.8 ± 3.4% (P < 0.001) in retinal veins.
In the schwannoma group, stimulation of the unaffected ear evoked significantly larger mean responses than the affected ear (Table 2; force: t(7) = 5.08, p = 0.001; velocity: t 9) = 3.23, p = 0.010; displacement: t 9) = 3.28, p = 0.010.
Therefore, for these more complex behaviors, the response to hemilineage group stimulation was probabilistic rather than deterministic, in that the activity of these neurons enhanced the probability that a particular behavior would be performed.
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As expected, a high percentage of both unstimulated and stimulated mLCs expressed CD1a in both age groups Stimulation did not significantly affect the level of expression of CD1a in either age group (Fig. 1a).
In order to test this hypothesis, we grouped stimulation phase favouring suppression or amplification according to proceeding phase values and whether these also favoured suppression or amplification.
Six small RNA libraries constructed from corresponding samples in PBS control group, ACh stimulation group and NE stimulation group were sequenced by Ion Torrent Proton.
Subjects were then randomized 1 1 to the Treatment group (active stimulation) or to the Sham group (no stimulation).
There was no difference in the SAE rate in the subjects in the Treatment group (active stimulation) and subjects in the Sham group (no stimulation).
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