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The results are consistent with Mudongo et al. (2016) who observed that continuous grazing led to diminished perennial grass population and increase in forbs and woody species in the Kalahari rangelands of Botswana.
(2008) showed that a restored dune grass population persisted with different genetic structure, possibly due to insufficient gene flow with natural populations or seedling recruitment.
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Similarly, a mixture of cyanazine + alachlor in cotton and atrazine + alachlor in maize is recommended for suppressing broad and grass weed populations and enhancing yields in CT systems.
Gaskin et al. (2012) observed minimum levels of genetic diversity in pepper grass weed populations (Lepidium latifolium) and suggested that they were due to founder effects or strong genetic bottlenecks before or after the dispersal events.
This is a common garden study that has four replications focused on differences in growth of 30 different barnyard grass populations that have been collected from 30 different sites all across the state of Texas.
Meadows are more stable and fertile environments, whose grass populations are older and well established by clonal spread.
Firstly, these results illustrate the importance of genetic incompatibility in out-crossing grass populations and maternal effects as destabilizing forces in endophyte-grass symbioses.
Namibian fountain grass populations are only found in human-derived disturbance areas such as roadsides with no spread or establishment in undisturbed native vegetation [8].
We quantified neutral and quantitative genetic diversity of globally invasive and native fountain grass populations to determine the importance of such variation in invasion success.
For example, seeds produced by outcrossing should have a high frequency of mismatches between the fungus and the grass and thus pioneer grass populations having a large portion of newly established individuals should have lower infection frequencies than older populations.
Our results demonstrate that genetic mismatches, maternal effects and loss of infections occur commonly in endophyte-grass interactions and may partly explain those differences detected in infection frequencies and genetic structures among natural grass populations [6], [36].
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