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TTH and migraine are separate headache disorders with different characteristics of GM change.
Notably, most migraine studies on GM change, despite heterogeneous findings in localization, have congruently demonstrated reduced GM in the pain processing network [10, 11, 13, 29 31].
Moreover, our findings cannot be generalized to the ictal imaging data because dynamic GM change across the ictal interictal cycle has been reported in TTH [20] and migraine [64].
A study on episodic TTH (<15 headache days/month) [20] did not reveal any <span class="lh">GM change, whereas a study on chronic TTH, as aforementioned, suggested reduced GM in the pain processing network [9].
Earlier MRI studies on TTH have revealed no volumetric GM change in episodic TTH [20] and reduced GM in chronic TTH in the anterior cingulate, insula, orbitofrontal cortex, dorsal pons, and other brain structures involved in pain processing [9].
However, the present findings of GM change in TTH and migraine (vs. controls) and the GM difference between both types of headaches mostly involved brain regions of the pain processing network, which suggested these plastic changes may reflect the allostatic load in response to headache pain [60].
Similar(51)
The stress in the channel region induced by the nitride layer changes transconductance (Gm), thereby changing the device performance.
Changes in cerebral blood flow may apparently change GM volume in VBM analyses [67].
Here is a picture: B1: Change GM to GM=Other.
Immediately after addition of Gm, we observed drastic change in the colloid color from wine red to purple blue.
Increased elevation also resulted in a decrease of SLA and Gm values, but no change in Gs, in the tropical Hawaiian tree species Meterosideros polymorpha (Cordell et al. 1998).
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