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Gist is based on one's culture, background and experiences, and experience is what teens lack".
Risk estimation of gastrointestinal stromal tumours (GIST) is based on tumour size and mitotic rate according to the National Institutes of Health consensus classification.
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The selection of patients with high-risk GIST was based on follow-up data from our previously published population-based study of 233 GIST patients (Nilsson et al, 2005).
Before 2002, the diagnosis of GIST was based on histology with variable use of staining markers.
The diagnosis of GIST was based on a combination of histopathological evaluation and immunohistochemistry for CD117.
The diagnosis of GIST was based on the routine histopathological examination and positive immunoreactivity for CD117 (KIT).
The diagnosis of GIST was based on the typical histological features of a cellular spindle cell or epithelioid mesenchymal tumour of the gastrointestinal tract.
Predictions about the biological behavior of GIST are based on a number of canonical clinical and pathologic parameters whose validity in distinguishing between a benign and a malignant tumour is still imperfect.
The reimbursement of GIST was based on the values set by the National Institute of Clinical Excellence (NICE) to determine the cost-effectiveness of sunitinib as compared to palliative therapy by utilising 5, 14, and 40% cuts of the highest NICE reimbursement level.
Diagnosis of GIST was based on currently applied diagnostic criteria [ 16, 22] using histological characteristics (e.g. highly cellular spindle/epithelioid/mixed cell tumors), immunohistochemical status (positivity for KIT or PDGFRα) and mutational analysis of relevant c-kit and PDGFRα exons.
The current diagnosis of GISTs is based on histological and immunohistochemical criteria, the most important of which is the expression of the receptor tyrosine kinase, KIT (CD117, c-Kit).
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