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To this end, we first address the mechanisms beyond normal adult neurogenesis, and secondly, the biochemical and genetic processes that drive cells towards tumor formation.
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Indeed, the increasing application of highly polymorphic genetic markers (such as microsatellites) is a recognition that defining population structure and population level genetic variation is critical to understanding the demographic processes that drive free-living protists distributions.
It is therefore important to monitor changes in the genetic and geographic complexity of the HIV-1 epidemic, and to identify the processes that drive these changes.
Coevolution is reciprocal evolutionary change in interacting species driven by natural selection, and is one of the most fundamental ecological and genetic processes that organize earth biodiversity [ 1, 2].
Such comparisons allow the detection and analysis of genetic processes that occur on relatively short timescales.
Little is known about the metabolic processes that drive the RNA silencing process.
A thought process that drives both people and progress.
We posit that the same process that drives speciation may also drive extinction.
Tumorigenesis in humans is a multistep process involving genetic alterations that drive the progressive transformation of normal cells into malignant types.
Instability of the genome is an important contributor to genetic changes that drive carcinogenic processes, and in accordance with the cancer field theory could provide a milieu where genetically altered cells would be more susceptible to the development of cancer [ 5, 24, 25].
Tumorigenesis is a multi-step process caused by genetic alterations that drive the progressive transformation of normal cells into malignant cells.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com