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We further assume that the host has genetically intrinsic capacities for growth and immune response [25], [38] hereafter simply called genetic growth and resistance potential, respectively.
This environmental sensitivity prior to birth may aid in preventing a mismatch between offspring genetic growth potential and maternal body size, which is the outcome of both genetics and past environment, and could raise the risk of obstructed labor (Wells, In press).
To address this issue, we developed several genetic growth switches based on CRISPR interference technology.
Growth defects overwhelmingly represent deficient rather than excess growth, and dozens of genetic growth failure syndromes have been identified.
Indeed, he said, recent discoveries show that the genetic growth signals often work by influencing cancer cells' metabolism.
In contrast, when nutrient availability is low the genetic growth potential is no longer the main driver of achieved growth.
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More recent studies have tried to link genetic of growth response to environmental variation [ 23, 32, 33].
Such interactions are classified in BioGRID as synthetic rescue, positive genetic, dosage growth defect, or dosage lethality.
Both are under complex genetic control: growth habit is controlled by the epistatic interactions of several genes.
Such interactions are classified in BioGRID as dosage rescue, negative genetic, synthetic growth defect, synthetic lethality, or synthetic haploinsufficiency.
Lipid accumulation increases throughout the adipogenic process, and it is regulated by genetic and growth factors [ 4].
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