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So it is not as if knowledge about genes will provide the full answer – at least, not until long into the future.
A better understanding of these genes will provide new insights into the disease pathogenesis.
aUPD may pinpoint regions with homozygous alterations and identifying those mutated genes will provide valuable insights.
The identification of these affected genes will provide more information about the function and virulence roles of virB.
Further investigation of these target genes will provide insights into the molecular basis by which Ac-DAF-16 regulates its downstream gene network in hookworm infection.
Further understanding of the induction of H3K4 trimethylation and increased expression of these genes will provide better insight into the epigenetic effects underlying the carcinogenicity of nickel compounds.
Further analysis of transferred genes will provide insight into the nature of the symbiotic relationship between Wolbachia and its filarial nematode hosts.
Discovery and confirmation of human longevity genes will provide insight into the biology of aging and the genetic basis for resistance to age-related disease.
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And because heredity only sets the stage for manic depression and schizophrenia, while environment opens the curtain, identification of even a central gene will provide limited answers.
Clarifying the compositions and effects of each gene will provide information about epistasis and effect of interactions of these genes.
Our on-going cloning effort of the Pi2-2 gene will provide us the answer in the near future.
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