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Expression changes in major pathogenesis-related genes were studied at different time courses.
Mutations in codon 209 of GNAQ and GNA11 genes were studied by restriction-fragment length polymorphism (RFLP).
In addition, the effects of salinity up-shock and nitrogen limitation on carotenoid content and expression of these two genes were studied in D. parva FACHB-815.
Three genes were studied in more details, 2 coding for metallothioneins (AgMT2 and AgMT3) and one for a new mannitol transporter (AgMaT3).
Hepatic ethoxyresorufin-O-deethylase (EROD) activity and hepatic expression of a number of aryl hydrocarbon receptor (AHR -regulated genes were studied to investigAHR -regulatedminated dioxins can activate genestranscription through the AHR pathwerein zebrafistudied
The enzymatic activity and expression of the three genes were studied.
The genes were studied using a network generated by PathwayStudioTM© software (Ariadne Genomics).
All other genes were studied using primers located in intronic regions, thus amplifying hnRNA and DNA.
Plasmid- and chromosome-encoded quinolone resistance determinants (qnr A, B and S, gyrA and parC genes) were studied [37], [38].
The tissue-level expression pattern of the AtCRT1a, CRT1b and CRT3 genes were studied using the β -glucuronidase (GUS) reporter gene.
46 genes were studied that had an experimental versus baseline expression (E/B) >2.0, based on the 90% confidence interval and E-B >50.
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