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These control genes were excluded from the candidates.
Therefore, these four genes were excluded as potential mediators of P2X7R-enhanced PDLSC osteogenesis under inflammatory conditions.
The second procedure, testing, employed a permutation test to evaluate whether possible genes were false discoveries; these genes were excluded by the testing procedure.
Our findings also support the proposal that screening of PRNP mutations should be performed for the patients with dominant ataxia if dynamic mutations of SCA genes were excluded.
Diseases having less than three seed candidate genes were excluded.
Intergenic regions between convergently-transcribed genes were excluded.
Consequently, the DRD2 and DYT1 genes were excluded with high confidence.
Pseudogenes, mitochondrial, plastid and insufficiently annotated genes were excluded from further analysis.
Start codons, stop codons, and overlapping regions of genes were excluded.
Predicted gene nodes without direct interactions to anchor genes were excluded from the network diagram.
Pathways containing over 300 or less than 2 genes were excluded.
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