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A total of 4,135 contigs representing putative single-copy genes were available for MIP design.
Expression data for 1,109 of the 1,168 grapevine PK genes were available, and the expression of these genes in the acquired datasets was analyzed at the PK family level (Supplementary Table S7).
For 656 of these patients, sequences of either viral gag, pol and nef genes were available.
Genotypes for 1536 SNPs in 357 cleft candidate genes were available from a previous analysis in which we searched for fetal gene effects in the same dataset [1].
Genotypes for 1536 SNPs in 357 cleft candidate genes were available from a previous analysis in which we focused on fetal gene effects [1].
After data-cleaning, genotypes for 1315 SNPs in 334 autosomal genes were available for the current analysis of maternal gene effects.
When duplicate measures of growth rate for strains with the same deleted genes were available, the average change in growth rate was used in our analysis.
Biological interpretations from single peptide hits were, however, made only when additional evidences such as similar dynamic profiles from functionally related genes were available.
After data-cleaning and exclusion of SNPs on the X-chromosome, genotypes for 1315 SNPs in 334 autosomal genes were available for the current analysis of maternal gene effects.
In these cases, not all cas genes were available.
Expression values for 3923 genes were available from this study.
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